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To a certain extent the dementias have been forgotten diseases until just recen tly when they were brought to the attention of the general public and health authorities as a result of the increasing number of cases in the aging popula tion, especially among famous people, and because of the efforts of private foundations. The goals of the present volume are to present the dementias to health prac titioners, to provide some basic information on their epidemiology and biolo gical basis and to discuss the diagnostic and clinical problems that physicians and institutions face when caring for demented patients. This book explores the various types of dementias and is not limited to Alzheimer's disease although, as expected, more information is available and presented on this pathology. On the other hand, a few fundamental questions on dementia can only be answe red through a comparison of the various forms. Examples of such questions are the following: Is the loss of cerebral tissue sufficient to cause dementia? Are there thresholds or is there a continuous progression toward the irreversible development of dementia? Are there common pathways in the dementing pro cess? Are there common risk factors? Comparative analysis allows the common and distinctive patterns of the various dementias to be defined, ultimately lea ding to more focused therapeutic interventions.
This book provides the first comprehensive overview of a new scientific discipline termed Geroscience. Geroscience examines the molecular and cellular mechanisms that might explain why aging is the main risk factor for most chronic diseases affecting the elderly population. Over the past few decades, researchers have made impressive progress in understanding the genetics, biology and physiology of aging. This book presents vital research that can help readers to better understand how aging is a critical malleable risk factor in most chronic diseases, which, in turn, could lead to interventions that can help increase a healthy lifespan, or ‘healthspan.’ The book begins with an analysis of the Geroscience hypothesis, as well as the epidemiological underpinnings that define aging as a candidate main risk factor for most chronic diseases. Next, each chapter focuses on one particular disease, or group of diseases, with an emphasis on how basic molecular and cellular biology might explain why aging is a major risk factor for it. Coverage in the book includes: cancer, cardiovascular disease, dementias, stroke, Parkinson's and Alzheimer’s diseases, osteoporosis, arthritis, diabetes asthma, emphysema, kidney disease, vision impairment, and AIDS/HIV. It finishes with a chapter on pain in the elderly and an overview of future steps needed to bring the newly acquired knowledge into the clinic and the public at large.
This volume contains chapters derived from a N. A. T. O. Advanced Study Institute held in June 1983. As the director of this A. S. I. it was my hope that some of the e1ectrophysiologists could express the potentialities of their work for perceptual theory, and that some perceptionists could speculate on the underlying "units" of perception in a way that would engage the imagination of physio logists. The reader will have to be the judge of whether this was achieved, or whether such a psychophysiological inter1ingua is still overly idealistic. It is clear that after the revolution prec~pitated by Hube1 and Weisel in understanding of visual cortical neurons we still have only a foggy idea of the behavioral output of any particular species of cortical detector. It was therefore particularly unfortunate that two persons who have made great strides in correlating interesting facets of cat cortical physio logy with human psychophysics (Max Cynader and Martin Regan of Dalhousie University) were unable to attend this meeting. Never theless, a number of new and challenging ideas regarding both spatial perception and cortical mechanisms are represented in this volume, and it is hoped that the reader will remember not only the individual demonstrations but the critical questions posed by the apposition of the two different collections of experimental facts. David Ingle April 1984 VII TABLE OF CONTENTS PREFACE V D. N. Lee and D. S. Young Visual Timing of Interceptive Action 1 J. J.