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Demand for agricultural crops and nutritional requirement continues to escalate in response to increasing population. Also, climate change exerts adverse effects on agriculture crop productivity. Plant researchers have, therefore, focused to identify the scientific approaches that minimize the negative impacts of climate change on agricultural crops. Thus, it is the need of the hour to expedite the process for improving stress tolerance mechanisms in agricultural crops against various environmental factors, in order to fulfil the world’s food demand. Among the various applied approaches, the application of phytohormones has gained significant attention in inducing stress tolerance mechanisms. Jasmonates are phytohormones with ubiquitous distribution among plants and generally considered to modulate many physiological events in higher plants such as defence responses, flowering and senescence. Also, jasmonates mediate plant responses to many biotic and abiotic stresses by triggering a transcriptional reprogramming that allows cells to cope with pathogens and stresses. Likewise, salicylates are important signal molecules for modulating plant responses to environmental stresses. Salicylic acid influences a range of diverse processes in plants, including seed germination, stomatal closure, ion uptake and transport, membrane permeability and photosynthetic and growth rate. Understanding the significant roles of these phytohormones in plant biology and from agriculture point of view, the current subject has recently attracted the attention of scientists from across the globe. Therefore, we bring forth a comprehensive book “Jasmonates and Salicylates Signalling in Plants” highlighting the various prospects involved in the current scenario. The book comprises chapters from diverse areas dealing with biotechnology, molecular biology, proteomics, genomics, metabolomics, etc. We are hopeful that this comprehensive book furnishes the requisite of all those who are working or have interest in this topic.
This book provides a comprehensive update on recent developments of Jasmonates (JAs) and Brassinosteroids (BRs) in plant signalling and biotechnological applications. Over the last few decades, an enormous amount of research data has been generated on these two signalling molecules. This valuable compilation will enhance the basic understanding of JAs and BRs mechanism of actions ensuing tolerance mechanism of crops under climate changes for sustainable agriculture and human welfare. This book covers topics regarding the occurrence of JAs and BRs in plants, biosynthesis, role in plant growth and development, role of these PGRs during various abiotic stress tolerance in plants, crosstalk of Reactive Oxygen Species (ROS) and plant stress mitigation, regulation of JAs and BRs signaling pathways by microRNA, along with physiological and anatomical roles of JAs and BRs as wound healing, regeneration and cell fate decisions. The cross talk of JAs and BRs with neurotransmitters in plant growth and development. Bio-fortification of crop plants with BRs in managing in human health issues chapter enlightened new role of BRs in human wellbeing. This book will be beneficial to scientists, researchers, agriculturists, horticulturists, industries related to the crop and food production KEY FEATURES Reviews the global scientific literature and experimental data of the authors on the occurrence of JAs and BRs in various plants Update information on recent developments of JAs and BRs signalling and biotechnological applications in plants Highlights the physiological, metabolic and molecular mechanism of JAs and BRs under variable climates Addresses the abiotic and biotic tolerance management by JAs and BRs Describes the role of JAs and BRs in sustainable agriculture and human welfare in eco-friendly manner
Rapid change in environmental conditions is likely to override the adaptive efficiency of plants.Jasmonates are a class of plant hormones that have strong potential to ameliorate various abiotic and biotic stresses, such as exposure to mechanical stress, osmotic stress, herbivory, exposure to UV irradiation, damage from ozone, and heavy metal stress. This new book highlights the beneficial presence and roles of jasmonates in plants, discussing the use of jasmonates in nano- and micromolecular concentrations to mitigate the toxic effects of various biotic and abiotic stresses and to improve crop yield in an eco-friendly and sustainable manner. It discusses in detail the genetic basis of jasmonates’ action, effects of jasmonates on the antioxidant defense system, crosstalk between jasmonates and other plant hormones, how jasmonates enhance plant defense under biotic and abiotic stresses, and more.
It is now well established that jasmonates, originally identified as the major component of jasmine scent, play a universal role in the plant kingdom and are involved in the regulation of diverse aspects of plant biology, including growth, development, metabolism, and interaction with the environment. In Jasmonate Signaling: Methods and Protocols, experts in the field aim to unite powerful emerging omics platforms with a number of key reductionist approaches to form a comprehensive collection of tools and protocols. The detailed chapters in this book embrace physiological, environmental, molecular, omics, and bioinformatics approaches that allow dissecting jasmonate actions in the model species Arabidopsis thaliana or in other plants. Written in the highly successful Methods in Molecular Biology series format, chapters feature introductions to their respective topics, lists of the necessary materials and reagents, step-by-step, readily reproducible laboratory protocols, along with tips on troubleshooting and avoiding known pitfalls. Authoritative and cutting-edge, Jasmonate Signaling: Methods and Protocols will empower interested researchers to dissect all steps of jasmonate signaling and the processes they modulate.
Systemic acquired resistance (SAR) develops in response to local microbial leaf inoculation and renders the whole plant more resistant to subsequent pathogen infection. Accumulation of salicylic acid (SA) in noninfected plant parts is required for SAR, and methyl salicylate (MeSA) and jasmonate (JA) are proposed to have critical roles during SAR long-distance signaling from inoculated to distant leaves. Here, we address the significance of MeSA and JA during SAR development in Arabidopsis thaliana. MeSA production increases in leaves inoculated with the SAR-inducing bacterial pathogen Pseudomonas syringae; however, most MeSA is emitted into the atmosphere, and only small amounts are retained. We show that in several Arabidopsis defense mutants, the abilities to produce MeSA and to establish SAR do not coincide. T-DNA insertion lines defective in expression of a pathogen-responsive SA methyltransferase gene are completely devoid of induced MeSA production but increase systemic SA levels and develop SAR upon local P. syringae inoculation. Therefore, MeSA is dispensable for SAR in Arabidopsis, and SA accumulation in distant leaves appears to occur by de novo synthesis via isochorismate synthase. We show that MeSA production induced by P. syringae depends on the JA pathway but that JA biosynthesis or downstream signaling is not required for SAR. In compatible interactions, MeSA production depends on the P. syringae virulence factor coronatine, suggesting that the phytopathogen uses coronatine-mediated volatilization of MeSA from leaves to attenuate the SA-based defense pathway.
The jasmonic (JA) and salicylic acid (SA) signaling systems regulate diverse plant developmental processes and immune responses. Due to the opposite end-goals of various pathogen-response pathways, a certain level of antagonism between the JA and SA pathways is required. However, over-suppression of any single pathway would leave a plant susceptible to invasion by a specific class of pathogen. We investigate the regulation of crosstalk between the JA- and SA-mediated immune pathways and how the dilemma of specific susceptibility may be avoided. Manipulation of Arabidopsis thaliana genotypes was utilized to determine the effects of protein knockout on pathogen-specific susceptibility and resistance. The effects of hormone treatment on protein concentration and the effects of protein knockout on the plant transcriptome were investigated as well. Loss of the plant U-box protein 25/26 increased susceptibility of plants to necrotrophic pathogen Pectobacterium carotovorum, indicating that this protein is instrumental in regulation of immune pathways required for optimization of fitness.
In der Induktion von Abwehrreaktionen in der Pflanze sind hauptsächlich die Pflanzenhormone Salizylsäure (SA), Jasmonsäure (JA) und Ethylen (ET) beteiligt.
Jasmonic acid (JA) is a plant hormone that controls many aspects of growth, development, and defense. As a defense hormone JA is rapidly synthesized in response to wounding, or attack from herbivores and pathogens, triggering the deployment of defense compounds. The activation of defenses by JA is accompanied by a potent repression of growth. This effect is thought to involve the diversion of resources from growth to defense. In this dissertation I investigated mechanisms regulating the plant wound response. In particular I performed genetic and biochemical studies on the JA-biosynthesis enzyme OPR3, which is implicated in the control of wound-induced JA biosynthesis. I found that OPR3 is chemically altered in wounded plants, and de-phosphorylation of an active site tyrosine hypothesized to control OPR3 activity is not necessary for OPR3 control in vivo. I also addressed the question of how JA signaling controls the balance between plant growth and defense. I performed a successful forward genetic screen to identify mutants that are insensitive to wound-induced growth inhibition. Seven mutants were identified which, unlike the wild-type, continue to grow during chronic wounding treatment. Four of these mutants maintain resistance to an insect herbivore and a necrotrophic pathogen suggesting that growth inhibition and the activation of defense responses can be uncoupled. I characterized one of the wound-insensitive mutants in detail. I show that the F-box protein, FBK51, is required for wound-induced growth inhibition, but not herbivore or pathogen resistance. I demonstrate that FBK51 interacts with the growth-promoting transcription factors VOZ1 and VOZ2, and the interaction leads to VOZ degradation. Transcriptional profiling of the wound response in fbk51 mutants suggests that a JA-independent pathway involving FBK51 may alter patterns of secondary metabolite accumulation and repression of photosynthesis. In summary, in this dissertation I address the question of how wounding triggers the rapid synthesis of JA, and how the wound response regulates the balance between plant growth and defense.