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Yield loss due to disease results from complex interactions between disease development and growth and development of plants. The effects of an Illinois isolate of wheat streak mosaic virus (WSMV) on yield of selected hard red spring wheat and soft red winter wheat cultivars were evaluated in three studies. In spring and fall of 1993 and 1994, twenty hard red spring wheat cultivars and fourteen soft winter wheat cultivars were planted in yield plots with three replications in a randomised complete block (RCB) design. Seedlings were mechanically inoculated at Feeke's growth stage 2 using an air brush. Controls were non-inoculated plots. Symptoms were rated every 7-8 days beginning one week after inoculation using a 0 to 5 scale, where 0 was no symptoms and 5 was severe stunting and chlorosis. The grain yield, 1,000 kernel weight and plant height were analyzed. There were significant differences among some cultivars for all the traits. Yield reductions in 1993 were greater than in 1994 which may have been caused by the differences in the environmental conditions. In the second study the response of three hard red spring wheat cultivars and four soft winter wheat cultivars when inoculated at different growth stages with WSMV were evaluated in yield plots in a RCB design with 3 replications. Seedlings were mechanically inoculated using an air brush at Feeke's growth stages of 2, 4, 6 and 8. Controls were noninoculated plots. Symptoms were rated as above. The response of the wheat cultivars to inoculation with WSMV at different growth stages was significant (Pr $>$ F = 0.0001) using repeated measures analysis. Analysis using ANOVA and FLSD mean separation showed significant differences in grain yield, 1000 kernel weight and height (P $
Many well-known specialists have contributed to this book which presents for the first time an in-depth look at the viruses, their satellites and the retrotransposons infecting (or occuring in) one plant family: the Poaceae (Gramineae). After molecular and biological descriptions of the viruses to species level, virus diseases are presented by crop: barley, maize, rice, rye, sorghum, sugarcane, triticales, wheats, forage, ornamental and lawn. A detailed index of the viruses and taxonomic lists will help readers in the search for information.
This book is a timely compilation of synthesized information on behaviourally fascinating and economically important mites. The book gives much attention to fundamental aspects of eriophyoid anatomy, behaviour, ecology and even systematics, as bases for understanding the ways of life of eriophyoid mites and their effects on host plants; in turn, this will lead to developing the most appropriate means of regulating mites as detrimental or beneficial organisms. It presents new views intended to stimulate interest in eriophyoids and their enemies, and it points to areas where further research is needed. This book is intended for extension workers, experts of acarology and plant protection as well as students, teachers and researchers. It stimulates readers to critically test the view presented and aimes ultimately toward environmentally safe, sustainable and economically efficient means of regulating detrimental and beneficial eriophyoid mites.
New viral diseases are emerging continuously. Viruses adapt to new environments at astounding rates. Genetic variability of viruses jeopardizes vaccine efficacy. For many viruses mutants resistant to antiviral agents or host immune responses arise readily, for example, with HIV and influenza. These variations are all of utmost importance for human and animal health as they have prevented us from controlling these epidemic pathogens. This book focuses on the mechanisms that viruses use to evolve, survive and cause disease in their hosts. Covering human, animal, plant and bacterial viruses, it provides both the basic foundations for the evolutionary dynamics of viruses and specific examples of emerging diseases. NEW - methods to establish relationships among viruses and the mechanisms that affect virus evolution UNIQUE - combines theoretical concepts in evolution with detailed analyses of the evolution of important virus groups SPECIFIC - Bacterial, plant, animal and human viruses are compared regarding their interation with their hosts
The wheat curl mite (WCM), Aceria tosichella Keifer, transmits a complex of viruses, Wheat streak mosaic virus (WSMV), Triticum mosaic virus (TriMV) and Wheat mosaic virus (WMoV), to wheat, Triticum aestivum, in the Great Plains. Co-infection of wheat by these viruses is frequently observed, increasing disease severity and yield loss. Current genetic work classifies WCM populations into two genotypes, Type 1 and Type 2. It has been shown that different mite genotypes are able to transmit viruses at varying rates. WCM-virus relations are very specific and can impact vector biology. In this study, the primary objective was to determine if co-infection of wheat by WSMV+ TriMV has an impact on each virus transmission rate by the WCM Type 1 and Type 2. An additional objective was to establish the impact of double viral infections on the biology of the mites and virus dispersal in the field. Using a series of transmission studies, it was determined that Type 1 WCMs do not transmit TriMV even in the presence of WSMV. Type 2 WCMs feeding on wheat infected with both viruses, have reduced WSMV transmission when compared to mites feeding on singly inoculated plants. However, TriMV transmission is increased when mites feed on wheat infected by both viruses. Mite counts from the field indicated that mites feeding on WSMV infected plants had the highest populations, followed by the control, WSMV+TriMV and TriMV. In field conditions, WSMV incidence was reduced when Type 2 WCM were exposed to source plants with WSMV+TriMV. TriMV incidence was not different between mites feeding on single or double infected plants. These findings enhance the understanding of WCM virus complex epidemiology.
Seeds provide an efficient means in disseminating plant virus and viroid diseases. The success of modern agriculture depends on pathogen free seed with high yielding character and in turn disease management. There is a serious scientific concern about the transmission of plant viruses sexually through seed and asexually through plant propagules. The present book provides the latest information along with the total list of seed transmitted virus and viroid diseases at global level including, the yield losses, diagnostic techniques, mechanism of seed transmission, epidemiology and virus disease management aspects. Additional information is also provided on the transmission of plant virus and virus-like diseases through vegetative propagules. It is also well known that seed transmitted viruses are introduced into new countries and continents during large-scale traffic movements through infected germplasm and plant propogules. The latest diagnostic molecular techniques in different virus-host combinations along with disease management measures have been included. The book shall be a good reference source and also a text book to the research scientists, teachers, students of plant pathology, agriculture, horticulture, life sciences, green house managers, professional entrepreneurs, persons involved in quarantines and seed companies. This book has several important features of seed transmitted virus diseases and is a good informative source and thus deserves a place in almost all university libraries, seed companies and research organizations.
Wheat viruses including Wheat streak mosaic virus (WSMV), Triticum mosaic virus (TriMV), High Plains wheat mosaic emaravirus (HPWMoV), and Barley yellow dwarf virus (BYDV) cost substantial losses in crop yields annually. Although there have been extensive studies conducted on known wheat viruses, currently, there is limited knowledge about all viral components associated with wheat (Triticum aestivum L.) including potential novel viruses. Wheat streak mosaic (WSM), a disease of cereals and grasses, costs Kansas farmers millions in yield losses. Although WSMV is considered as the main causal agent of WSM, TriMV and HPWMoV have also been reported in mixed infections. While resistant varieties are utilized to minimize the effects of the disease, genetic variation in associated viruses increases the emergence of potential resistance-breaking isolates. Currently, little is known about the genetic composition of populations of WSM-associated viruses in the field. This study first aims to analyze the genetic variation and characterize the evolutionary mechanism(s) applied by WSM-associated viruses in the field using complete genomes sequences, and also to determine and characterize all viral populations associated with wheat. Field collections of 24 WSM-like symptomatic and asymptomatic wheat samples were used for total RNA deep sequencing, along with 5 historic WSMV samples for the evolutionary studies. Through bioinformatics analysis, sequences were mapped to available reference genomes and de novo assembled to identify new viruses. Results of the 2019 field survey showed WSMV as the predominant virus followed by mixed infections of WSMV+TriMV. Recombination was observed to be a major evolutionary force for WSMV but not for TriMV isolates. Phylogenetic analyses based on the obtained full genome sequences demonstrated that, unlike other isolates from the United States, Kansas isolates are widely distributed in sub-clades. Moreover, the phylogenetic studies suggested that TriMV field isolates may be under selection pressure to introduce genetic variations due to the use of resistant varieties in the fields. The full genome sequence of a new Kansas HPWMoV isolate was reported here. In addition to known wheat viruses, viral sequences sharing significantly low (
Viral hemorrhagic fevers (VHFs) are a group of illnesses that are caused by several distinct families of viruses. While some types of hemorrhagic fever viruses can cause relatively mild illnesses, many of these viruses cause severe life-threatening disease. Some examples include: Lassa fever, Marburg virus, Ebola virus, Bolivian hemorrhagic fever, Korean hemorrhagic fever, Crimean-Congo hemorrhagic fever and Dengue hemorrhagic fever. No current treatment can cure viral hemorrhagic fevers, and immunizations exist for only two (yellow fever and Argentine hemorrhagic fever) of the many VHFs. Researchers are working to develop other vaccines, but in the meantime, the best approach is prevention. This volume will provide a review of what is known to date on these virus families as well as highlighting recent advances and future needs. Key features: * Provides comprehensive overview of what is known to date, recent advances and future needs * Examines transmission and risk factors * Highlights what has been done to help in outbreak control * Discusses the need for vaccines and antivirals