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With high morbidity and mortality rates, atherosclerosis (ATS) is a major concern worldwide. This book provides comprehensive information on ATS, including chapters on cardiovascular disease and obstructive sleep apnea, rheumatology and cardiovascular risk, and the differences of ATS among men and women. Let’s hope for the best and thank you to all my team for collaborating in this book production. In particular, thanks • to Bruno (my expert colleague) and Aurel (my secretary) for analyzing and reading with me all the texts • to Vincenzo, Stefano and Andrea for English evaluation • and finally to Rebecca (my future wife) for sustaining me in all professional activities giving me power in daily life!
The causes, evolution, prevention, control, and treatment of arteriosclerosis are the focus of this report of the Working Group on Arteriosclerosis. Current knowledge of the pathogenesis, clinical manifestations, and risk factors associated with atherosclerosis are assessed. Conclusions and recommendations of the working group are presented in the areas of fundamental research, prevention, biobehavioral research, and health research personnel.
Selected as an outstanding book in vascular surgery by members of the Society for Vascular Surgery.* This unique new text describes the current understanding of the etiology and pathogenesis of human atherosclerosis. It also details the methods for quantitating and characterizing both experimental and clinical lesions, and describes the methods for preparing available animal models. Providing an in-depth review of each of these topics, the text organizes the information in one volume for the convenience of the reader. The text is divided into two sections. First is a description of the cell biology, biochemistry and pharmacology of normal vessels and of atherosclerotic human lesions, with details of the methods to accurately characterize and quantitate the disease. Secondly, it presents a description of the methods for preparing the available experimental animal models, including a discussion of the distribution and pathological characteristics of the lesions. It also includes comparisons of human atherosclerosis and experimental animal models. Intended to provide a basis for expediting future research in this priority health care area, this text compiles the available information for those who treat patients with atherosclerosis or who are involved in atherosclerosis research. It is of particular interest to students, physicians, and academic and commercial researchers.

Arteriosclerosis

1977 Working Group to Review the 1971 Report by the National Heart and Lung Institute Task Force on Arteriosclerosis

Published: 1977

Total Pages: 116


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New updated edition first published with Cambridge University Press. This new edition includes 29 chapters on topics as diverse as pathophysiology of atherosclerosis, vascular haemodynamics, haemostasis, thrombophilia and post-amputation pain syndromes.
Substantial progress toward reconciling disparate theories of the pathogenesis of arteriosclerosis was recorded in Volume I of this book. the proceedings of an interdisciplinary workshop conference held at Lindau. Germany, April 19-25, 1970. Strong evidence was ad duced that the early stages of arteriosclerosis consist of intimal proliferation similar to that associated with the increase in arteri al size and caliber that characterizes normal growth. The findings described recall a theory proposed by Richard Thoma (Thoma and Kaefer, 1889) of Heidelberg toward the end of the nineteenth century. He sug gested that the pathogenesis of arteriosclerosis began with intimal thickening that was initially adaptive. Past a certain point, how ever, the thickening compromised the nutrition of the artery wall, leading inevitably to degenerative changes. In 1944 Hueper (Hueper, 1944), reemphasized the threat to the nu tritional support of the artery and proposed that the many etiologic factors capable of inducing arterial atheroma did so through the final common pathway of interfering with oxidative mechanisms in the vessel wall. Thus, it is proposed that arteriosclerosis is basically an aspect of the "behavior" of the artery. The nature of the lesion, therefore, depends more on the biochemical and structural response capabilities of the tissue itself than on the characteristics of the various etiologic agents. The idea that arteriosclerosis begins as uncontrolled or disturbed adaptive behavior was shared by Duff and also by Winternitz (Duff, 1954; Winternitz, 1954).